What is it? A distinctive feature of this disease is bone fragility. Weak bones due to a decrease in calcium absorption in the body and depletion of bone calcium and protein. This is the most common type of metabolic bone disease in which bones become thinner and weaker, as part of aging. Bones may be broken and heal slowly, occurring especially in women after menopause. If left untreated, skeletal bones become fragile and some are likely to crack or fracture, especially hip and vertebrae. Osteoporosis is most common in women over the age of 50 years. After menopause, women experience rapid bone loss due to reduction in the production of estrogen. Any bone can be broken, but special concern cause fractures of the hip and spine.
Back and spine fractures can cause loss of height, severe back pain, nerve compression and deformation. The following risk factors associated with osteoporosis: smoking, chronic disease, alcohol use, high caffeine intake, early hysterectomy with ovarian removal appropriate, hyperthyroidism, and chronic steroid use drugs. Vitamin D plays an important role in calcium absorption. Creating strong bones, eating calcium rich foods, support balanced diet and exercise at an early age may be the best protection against osteoporosis. Image used with permission of Medtronic These images show osteoporosis. The upper image shows a fracture of the thoracic spine. This type of fracture is called a compression fracture. Note the wedge shape of the vertebrae, as he fell to the front. The resulting image shows a normal left vertebral osteoporosis and vertebrae on the right. Note the loss of bone in the body of vertebra with a few fragments of bones left. What are the symptoms? Loss of bone mass occurs without symptoms. People can lose bone mass over a long period of time without realizing it. The first signs of osteoporosis >> << frequently fractured spine, hip or wrist after a simple fall. When bone loss is a serious and osteoporosis is high, the following symptoms >> << height loss and spine slouch main effects osteoporosis fractures of the spine. How to diagnose? Osteoporosis is diagnosed spinal X-rays, spinal CT scan or bone density (DEXA scan). Suspicion of osteoporosis is usually present when there is fracture of the spine where there is only a small amount of this effect, such as a simple fall. How is it treated? The best treatment is prevention. Adequate calcium and vitamin D intake and exercise are important preventive tools against osteoporosis. Avoid smoking and excessive alcohol consumption and other important factors. There are several medications that can help increase bone density. Antiresorptive drugs, biphosphonates (alendronate, and ibandronat ryzedronat), calcitonin, estrogen and raloksifen affect bone remodeling cycle. Teriparatide, a form of parathyroid hormone, is a newly approved drug against osteoporosis. This is the first drug to speed the formation of bone in bone remodeling cycle. Fractures that are supported by osteoporosis can be treated with Kifoplastika, vertebroplasty, bed or external mounting. Kifoplastika a procedure in which a balloon inserted into the vertebra raise the collapsed segments and then bone cement is introduced into the space created by balloon. In vertebroplasty, bone cement is injected into the spine, not a balloon inflated. These procedures are performed with needles inserted through the skin on the back (see
Kifoplastika and vertebroplasty). External mounting requires that the patient wears braces. Does this treatment is the complication? There is little risk associated with vertebroplasty and Kifoplastika that bone cement may leak lasix 50 mg iv into the blood vessels or in places where the nerves. This is a rare and avoided careful surgical technique. Talk to your specialist about other risks (see Your Back Operation). What are the long-term expectations? Osteoporosis is a progressive disease that can lead to fractures of the current that can be disabled. Prevention best advice, but there is hope of new procedures that are available. It is important to diagnose osteoporosis as early as possible. .

Adhesion molecules, which determine the localization of naive lymphocyte trafficking in lymph nodes. Ligands HEU for guidance lymphocyte receptors
The immune system is largely goalkeeper for your body. It keeps the good things in and protect you from bad things. The immune system can be fairly complex topic, the following information to clarify some confusion on this issue. The immune system is one of the main biological systems that everyone has. It is rather unique in that the immune system does not exist primarily in one location, and more or less throughout the body. For example, respiratory system consists of lungs, diaphragm, trachea and other organs used for breathing. Skeletal system consists of all the bones that are in your body. In contrast, the immune system does not exist in one place, and is a collection of systems and protection mechanisms that are designed to keep people safe. The immune system must be organized, as it is because although most infections are making their way into the body through various holes in the head, most diseases and pathogens are much more complex actions than simply find their way into the body through open holes. Primary immunodeficiency immune system is the greatest defenses against disease, and unfortunately, there are many people in the world who happen to be >> << weakened immune system (often referred to as HIV). There are two main ways in which people can get weakened immune system. First, it is almost always genetic and occurs when a person is born is called
Article reviewed Crohn Anita updated: March 8, 2011
When using live patients and lightweight designs, they can learn the light at two different levels of permission. For patients with COPD, which are high resolution, CT, the authors quantified the number of small airways (defined as the measurement of respiratory tract 2-2. 5 mm internal diameter). They then used various kinds of high-resolution CT, which can not be used in clinical settings to get even closer view of the pulmonary parenchyma, this time donated samples of light. Lung samples were frozen, and the authors were able to attend the branching of the smallest airways to calculate the number and diameter of terminal bronchioles. What they found that patients with COPD were less worse and have small airways. Indeed, eksplantirovannyh lungs (in patients with emphysema is transplant) was 72 to 89% less terminal bronchioles (bronchioles that do not end in alveoli) than controls. to patients with less severe COPD also had less and have smaller airways. While it was not possible to determine whether there is a small loss of airway prior to emphysematous destruction of alveolar air space, the authors hypothesized that small airways were actually lost in the first place. It should be noted that the authors recognize that itBЂ ™ s possible CT was not sufficiently sensitive instrument, and the airways were not removed as a whole, but it really comes down to the gap is too small to be noticed CT. In any case, functional impairment, most likely the same. They concluded: BЂњThere widespread narrowing and loss of lower respiratory tract before the emphysematous destructionBЂ | This process readily explains the observed increaseBЂ | in small airway resistance in patients with COPD. BЂ "